Shoulder Injury Assessment Quiz
Shoulder Injury Assessment Quiz
Test your knowledge on shoulder injuries with our comprehensive quiz designed for healthcare professionals and students. Explore real-world scenarios, injury classifications, and clinical features associated with various shoulder-related conditions.
- 16 carefully crafted multiple-choice questions
- Focus on assessment of AC joint injuries
- Explore pathology related to shoulder dislocation and impingement
Result in the complete disruption of the AC and coracoclavicular ligaments, but the deltotrapezial fascia remains intact. Radiographs reveal a 25% to 100% increase in the coracoclavicular interspace relative to the normal shoulder.
Type 2
Type 3
Type 4
Type 5
Involve complete disruption of the coracoclavicular and AC ligaments, with posterior displacement of the distal clavicle into the trapezius muscle.
Type 2
Type 3
Type 4
Type 5
Involve the complete disruption of the AC ligaments but with intact coracoclavicular ligaments. Radiographs might demonstrate clavicular elevation relative to the acromion but less than 25% displacement.
Type 2
Type 3
Type 4
Type 5
Involve a mild injury to the AC ligaments, and radiologic evaluation is normal.
Type 1
Type 2
Type 3
Type 4
Involve complete disruption of the coracoclavicular and AC ligaments, as well as the deltotrapezial muscular attachments, with displacement of the distal clavicle below the acromion or the coracoid process.
Type 3
Type 2
Type 4
Type 6
The coracoclavicular and AC ligaments are fully disrupted along with a rupture of the deltotrapezial fascia. This results in an increase in the coracoclavicular interspace to greater than 100% of the normal shoulder.
Type 3
Type 4
Type 5
Type 6
This injury is caused by repeated anterior shoulder subluxations. The dislocation of the shoulder joint (anterior) can damage the connective tissue ring around the glenoid labrum.
Bankart lesion
Reverse Bankart lesion
Hill-Sachs lesion
Reverse Hill-Sachs lesion
Is defined as the detachment of posteroinferior labrum with avulsion of posterior capsular periosteum.
Bankart lesion
Reverse Bankart lesion
Hill-Sachs lesion
Reverse Hill-Sachs lesion
Is a compression fracture or "dent" of the posterosuperolateral humeral head that occurs in association with anterior instability or dislocation of the glenohumeral joint.
Bankart lesion
Reverse Bankart lesion
Hill-Sachs lesion
Reverse Hill-Sachs lesion
Also called a McLaughlin lesion, is defined as an impaction fracture of anteromedial aspect of the humeral head following posterior dislocation of the humerus.
Bankart lesion
Reverse Bankart lesion
Hill-Sachs lesion
Reverse Hill-Sachs lesion
Occurs for the first 1 to 3 months and involves pain with shoulder movements but no significant glenohumeral joint ROM restriction when examined under anesthesia.
Stage 1
Stage 2
Stage 3
Stage 4
The “freezing stage,” symptoms have been present for 3 to 9 months and are characterized by pain with shoulder motion and progressive glenohumeral joint ROM restriction in forward flexion, abduction, and internal and external rotation.
Stage 1
Stage 2
Stage 3
Stage 4
The “frozen stage,” symptoms have been present for 9 to 15 months and include a significant reduction in pain but maintenance of the restricted glenohumeral joint ROM.
Stage 1
Stage 2
Stage 3
Stage 4
Frequently referred to as the “thawing stage,” symptoms have been present for approximately 15 to 24 months and ROM gradually improves.
Stage 1
Stage 2
Stage 3
Stage 4
Hooked acromion or a thick coracoacromial ligament.
Primary impingement
Secondary impingement
Has many causes, including glenohumeral joint instability, weak scapular stabilizers, scapulothoracic dyskinesis, and instability.
Primary impingement
Secondary impingement
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